Idiopathic generalized epilepsy represents about one third of all epilepsy disorders, and there is increasing evidence that genetic factors play an important role in its development [1]. Considerable progress has been made over recent years in identifying genes that contribute to monogenic forms of epilepsy. The genes identified are components of neuronal signalling, which is composed of voltage-gated ion channels, neurotransmitter receptors, ion channel associated proteins and synaptic proteins [2].

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