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Proteome-wide Mendelian Randomization and Colocalization Nominate CD58, PARP1, and Immune Co-stimulatory Pathways as Genetically Supported Targets for Multiple Sclerosis

Study Overview The research focused on exploring the genetic underpinnings of multiple sclerosis (MS) through advanced analytical methods, specifically Mendelian

Proteome-wide Mendelian Randomization and Colocalization Nominate CD58, PARP1, and Immune Co-stimulatory Pathways as Genetically Supported Targets for Multiple Sclerosis Read Post »

Stem cell-derived extracellular vesicles as immunomodulatory agents: targeting pathological crosstalk in systemic lupus erythematosus and multiple sclerosis

Pathophysiology of Systemic Lupus Erythematosus and Multiple Sclerosis Systemic lupus erythematosus (SLE) and multiple sclerosis (MS) are both chronic autoimmune

Stem cell-derived extracellular vesicles as immunomodulatory agents: targeting pathological crosstalk in systemic lupus erythematosus and multiple sclerosis Read Post »

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